“Multiple System Atrophies”  

Multiple System Atrophy

    In our body, multiple system atrophy is a group of progressive neurodegenerative dis-orders, involving complex autonomic nervous system dysfunctions indicative to a specific set of signs, symptoms or other health indicators, associated with basal ganglia dis-orders and movement dis-orders.
    In our body's central nervous system, Multiple System Atrophy with primary dysautonomia symptoms that reflect the loss of nerve cells in several different areas in the brain and spinal cord that control the autonomic nervous system and coordinate muscle movements
    Examples of multiple system atrophy related and/or associated dis-orders include:

• Olivopontocerebellar Atrophies
• Shy-Drager Syndrome
• striatal degeneration

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“Pantothenate Kinase-Associated Neurodegeneration (PKAN)”  

PKAN (Hallervorden-Spatz Disease)

    In our body's brain, the basal ganglia related dis-order, Pantothenate Kinase-Associated Neurodegeneration (PKAN), AKA Hallervorden-Spatz disease, is indicative to a specific set of signs, symptoms or other health indicators, associated with a neuroaxonal dystrophy health challenge.
    In our body, Pantothenate Kinase-Associated Neurodegeneration (PKAN) is a heredodegenerative dis-order of the brain, accompanied by an excess of iron that progressively builds up in the brain, that can lead to movement dis-orders, dystonia, parkinsonism, dementia, and ultimately death.

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“Huntington's Disease (HD)”  

Huntington Disease

    In our body, Huntington's Disease is a nervous system heredodegenerative dis-order in which nerve cells in the striatum (neostriatum) of the brain waste away, or degenerate caused by a defect on chromosome 4.

The CAG section of the DNA, normally is repeated 10 to 28 times, but in persons with HD, it is repeated 36 to 120 times.
    In our body, Huntington's Disease is an adult-onset dominant heritable basal ganglia dis-ease characterized by progressive psychiatric disruption, cognitive deficits, which can lead to dementia, and loss of motor coordination (choreoathetosis).
    In our body, Huntington's Disease is caused by an abnormal elongation of a polyglutamine (polyQ) tract within the N-terminal domain of the Huntingtin protein, encoded by HTT gene involved with the HTT-AS1 gene.

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“Progressive Supranuclear Palsy”  

    In our body's brain, the neurodegenerative dis-order, Progressive Supranuclear Palsy is a palsy indicative to a specific set of signs, symptoms or other health indicators, associated with ophthalmoplegia and tauopathies.
    In our body, Progressive Supranuclear Palsy as a basal ganglia dis-order, affects cognition, eye movements and posture, with characteristics that including supranuclear, primarily vertical, gaze dysfunction accompanied by extrapyramidal symptoms and cognitive dys-functions.

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“Tourette Syndrome (Tourette's Disorder)”  

Tourette Syndrome

    In our body, the neuropsychological dis-order, Tourette Syndrome (Tourette's Disorder), aka Gilles De La Tourette's Syndrome, is a complex tic disorder, associated with multiple tics, involving both motor and phonic tics, related to alterations in dopamine metabolism and neurotransmission involving frontal-subcortical neuronal circuits.
    In our body, Tourette Syndrome (Tourette's Disorder) is a heredodegenerative dis-order, indicative to a specific set of signs, symptoms or other health indicators, associated with a movement disorder health challenge.

    In our body, a Tourette Syndrome is an infectious associated disease linked or correlated with at least one infectious pathogen.

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“Chorea Gravidarum (Pregnancy-Associated Choreoathetosis)”  

Chorea Gravidarum

    In a pregnant woman's body, the rare pregnancy complication of Chorea Gravidarum (Pregnancy-Associated Choreoathetosis) is a choreoathetosis basal ganglia movement dis-order developed during the pregnancy, characterized by involuntary jerky motions (chorea) and an inability to maintain stable position of body parts (athetosis).

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“Hepatolenticular Degeneration (Wilson's Disease)”  

Hepatolenticular Degeneration

    In our body's cells, the autosomal recessive genetic liver dis-order, Hepatolenticular Degeneration (Wilson's Disease) involves a mutation of the gene coding for the copper-transporting ATPase 2, which prevents copper within the liver from entering the Golgi apparatus in cells, characterized by the deposition of copper in the brain, liver, cornea and other organs.

Wilson's Disease (Hepatolenticular Degeneration)

    In our body, Hepatolenticular Degeneration is a heredodegenerative dis-order of copper metabolism caused by defects in the ATP7B gene encoding copper-transporting ATPase 2 (Wilson disease protein). The overload of copper inevitably leads to progressive liver and neurological dysfunction such as liver cirrhosis, tremors, ataxia and intellectual deterioration. Hepatic dysfunction may precede neurologic dysfunction by several years.

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“Neuroleptic Malignant Syndrome”  

    In our body, Neuroleptic Malignant Syndrome is a dis-order, indicative to a specific set of signs, symptoms or other health indicators, associated with a health challenge.

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“Meige Syndrome”  

    In our body, Meige Syndrome is the allopathic name of a dis-order, indicative to a specific set of signs, symptoms or other health indicators, associated with a health challenge.

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